By Dolores G. Evans, Francisco F. J. de la Cabada, Doyle J. Evans Jr. (auth.), S. Kuwahara, N. F. Pierce (eds.)
The United States-Japan Cooperative scientific technology software used to be initiated in 1965 via joint contract among the President of the U.S. and the top Minister of Japan. the aim of this system was once to advertise cooperative biomedical learn among the 2 nations, specially on illnesses of well-known impor tance in Asia. Cholera was once particular as one subject of mutual curiosity. Panels of scientists from each one state have been shaped, and those met to pick precedence components for learn. The Cholera Panels before everything outlined significant targets: 1) more suitable and simplified treatment for cholera, and a pair of) larger tools for immunization. development within the pursuit of those objectives resulted in the popularity that micro organism except Vibrio cholerae also are very important reasons of acute dehydrating diarrhea which resembles cholera in its manifestations and patho genesis; such a lot awesome between those are enterotoxinogenic lines of Escherichia coli. as a result, panel instructions have been accelerated to incorporate all diarrheal ailments that contain fluid loss because of an enterotoxin. extra lately, experiences have proven that vibrios, together with V. cholerae, have a special environmental lifestyles cycle that's most likely an impor tant think about the epidemiology of vibrio infections. as a result, the panel directions have been back multiplied to incorporate stories at the environmental ecology of vibrios. a massive undertaking of the Joint Cholera Panels has been the association and spon sorship of an annual convention on cholera and comparable diarrheal diseases.
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Additional resources for Advances in Research on Cholera and Related Diarrheas
1978. The role of antigen form and function in the primary and secondary intestinal immune responses to cholera toxin and toxoid in rats. J. Exp. Med. 148: 195-206. 5. Fuhrman, J. , Cebra, J. J. 1981. Special features of the priming process for a secretory IgA response. B cell priming with cholera toxin. J. Exp. Med. 153:534-544. 6. Pierce, N. , Gowans, J. L. 1975. Cellular kinetics of the intestinal immune response to cholera toxoid in rats. J. Exp. Med. 142:1550-1563. 7. , Holmgren, J. 1976. Synergistic protective effect in rabbits of immunization with Vibrio cholerae lipopolysaccharide and toxin/toxoid.
D. F. Jackson. 1981. Immune responses of rats to live Vibrio cholerae: Antibodies in serum and intestinal secretions. Parasite Immunology 3, 57-68. , S. Kuwahara, N. F. Pierce, 29-33. Copyright © 1983 by KTK Scientific Publishers, Tokyo. PROTECTION OF NEONATAL PIGLETS AGAINST COLIBACILLOSIS BY IMMUNIZATION OF DAMS WITH PROCHOLERAGENOID E. J. , F. Dorner and R. Germanier Swiss Serum and Vaccine Institute Berne Lethal Escherichia coli-induced diarrhea (enteric colibacillosis) results in major worldwide economic losses (8).
There are several possible explanations for these differences. But whatever the actual explanation, it is apparent that B subunit is markedly inferior to CT as an oral immunogen, at least in this animal model. Further studies with this antigen have not been pursued . Oral Immunization with Heat Aggregated Cholera Toxin: The final series of studies in this report involved evaluation of heat aggregated cholera toxin (procholeragenoid; abbreviated HACT) as a possible oral immunogen. Earlier studies by Fujita and Finkelstein and by Peterson have suggested that HACT is immunogenic given orally (10, 11).